51-267
Fisiopatología de la necrosis de la cabeza femoral no traumática. Estado actual de conocimientos.
Pathophysiology of non-traumatic necrosis of femoral head. Current state of knowlegde.
I. Capó Soliveres.
Recepción:
22/11/2016
Aceptación:
22/11/2016
Publicación:
22/11/2016
Métricas
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Resumen (ES)
Necrosis of the femoral head in the atraumatic form affects a young population, so the efforts of the researchers are aimed at clarifying the aetiology and pathophysiology of this disease, to establish the basis of prevention and non-surgical treatment; thus attempting to avoid reaching the phases of femoral head collapse and arthroscopic degeneration of the hip joint, which necessitate placing a total replacement arthroplasty at an early age. Alcoholism, coagulopathies, sickle cell disease and HIV infection are the aetiologies described in more detail, although all of them reach a common pathway of ischemia and bone necrosis, a process of repair in favor of bone resorption with loss of Structural integrity and subchondral collapse. All this favored by a genetic predisposition. As for treatment, there is a tendency to preserve the replacement arthroplasty for subchondral collapse and to attempt more conservative alternatives in pre-collapse stages, although the best results are obtained with arthroplasty.
Palabras clave (ES):
OsteonecrosisCaderaFisiopatologíaCabeza
femoral
Resumen (EN)
Necrosis of the femoral head in the atraumatic form affects a young population, so the efforts of the researchers are aimed at clarifying the aetiology and pathophysiology of this disease, to establish the basis of prevention and non-surgical treatment; thus attempting to avoid reaching the phases of femoral head collapse and arthroscopic degeneration of the hip joint, which necessitate placing a total replacement arthroplasty at an early age. Alcoholism, coagulopathies, sickle cell disease and HIV infection are the aetiologies described in more detail, although all of them reach a common pathway of ischemia and bone necrosis, a process of repair in favor of bone resorption with loss of Structural integrity and subchondral collapse. All this favored by a genetic predisposition. As for treatment, there is a tendency to preserve the replacement arthroplasty for subchondral collapse and to attempt more conservative alternatives in pre-collapse stages, although the best results are obtained with arthroplasty.
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