34-199
Síndrome de Parsonage-Turner. Diagnóstico y tratamiento.
Parsonage-Turner syndrome. Clinical diagnosis and treatment.
M. LÓPEZ ESTEVE, A. QUERCHFELD BELTRAMI, R. GÓMEZ GÓMEZ, M. A. CHAPA VILLALBA SERVICIO DE REHABILITACIÓN. HOSPITAL CLÍNICO UNIVERSITARIO DE VALENCIA.
Recepción:
15/09/2008
Aceptación:
15/09/2008
Publicación:
15/09/2008
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Resumen (ES)
Six patients who had acute brachial neuritis
(Parsonage-Turner Syndrome) were retrospectively studied. All
patients had an acute onset of intense pain that decreased spontaneously,
followed by weakness of the shoulder.
Electromyography revealed peripheral neuromyogenic lesion
without evidence of radicular distribution or affectation of individual
peripheral nerve. The overall prognosis was excellent despite
the severity and extension of the lesion. Pareses slowly regressed
over the following months with physiotherapy.
(Parsonage-Turner Syndrome) were retrospectively studied. All
patients had an acute onset of intense pain that decreased spontaneously,
followed by weakness of the shoulder.
Electromyography revealed peripheral neuromyogenic lesion
without evidence of radicular distribution or affectation of individual
peripheral nerve. The overall prognosis was excellent despite
the severity and extension of the lesion. Pareses slowly regressed
over the following months with physiotherapy.
Palabras clave (ES):
Hombro
Doloroso
Tendinitis
Neuritis
Parsonage-Turner
Síndrome
Diagnóstico
Tratamiento
Resumen (EN)
Six patients who had acute brachial neuritis
(Parsonage-Turner Syndrome) were retrospectively studied. All
patients had an acute onset of intense pain that decreased spontaneously,
followed by weakness of the shoulder.
Electromyography revealed peripheral neuromyogenic lesion
without evidence of radicular distribution or affectation of individual
peripheral nerve. The overall prognosis was excellent despite
the severity and extension of the lesion. Pareses slowly regressed
over the following months with physiotherapy.
(Parsonage-Turner Syndrome) were retrospectively studied. All
patients had an acute onset of intense pain that decreased spontaneously,
followed by weakness of the shoulder.
Electromyography revealed peripheral neuromyogenic lesion
without evidence of radicular distribution or affectation of individual
peripheral nerve. The overall prognosis was excellent despite
the severity and extension of the lesion. Pareses slowly regressed
over the following months with physiotherapy.
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