28-164
Escoliosis experimental por lesión vascular metamérica a nivel lumbar
EXPERIMENTAL SCOLIOSIS BY VASCULAR DAMAGE OF THE NEUROCENTRAL CARTILAGE AT THE LUMBAR SPINE
A. A. VALVERDE NAVARRO, M. MIQUEL FEUCHT, J. L. BERLANGA HERNANDEZ, J. BAEZA NOCI, M. MARTI FAUS y T HERNANDEZ GIL DE TEJADA Unidad de Raquis Experimental. Departamento de Ciencias Morfológicas. Facultad de Medicina y Odontología. Universidad de Valencia.
Recepción:
13/01/2009
Aceptación:
13/01/2009
Publicación:
13/01/2009
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Resumen (ES)
We have damaged the vascular supply to the right neurocentral cartilage (NCC)to 25 growing rabbits, in order to induce scoliosis. The employed technique was the sectionof the right metameric artery and vein at two or three levels in the lumbar region (L3 to L5).The spine was studied histologically macroscopically and radiologically. We got slight curves(13 ± 4 ° ) with the convexity towards the opposite side to the operated zone, and with rotation(12 ± 5 ° ) and lordosis (5 ± 7°). These lesions are similar to human idiopathic scoliosis and maybe explained because of the NCC's physiological properties. Those findings support the ideathat any mechanic, metabolic or endocrine alteration that cause damage to the NCC or to itsvascularization, unilaterally, will induced a scoliosis development.
Palabras clave (ES):
Idiopática
Escoliosis
Experimental
Lesión
Metamérica
Vascular
Lumbar
Etiología
Fisiología
Resumen (EN)
We have damaged the vascular supply to the right neurocentral cartilage (NCC)to 25 growing rabbits, in order to induce scoliosis. The employed technique was the sectionof the right metameric artery and vein at two or three levels in the lumbar region (L3 to L5).The spine was studied histologically macroscopically and radiologically. We got slight curves(13 ± 4 ° ) with the convexity towards the opposite side to the operated zone, and with rotation(12 ± 5 ° ) and lordosis (5 ± 7°). These lesions are similar to human idiopathic scoliosis and maybe explained because of the NCC's physiological properties. Those findings support the ideathat any mechanic, metabolic or endocrine alteration that cause damage to the NCC or to itsvascularization, unilaterally, will induced a scoliosis development.
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